Lanthanum chloride inhibition of the secretory response to Escherichia coli heat-stable enterotoxin

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Lanthanum chloride inhibition of the secretory response to Escherichia coli heat-stable enterotoxin.

Escherichia coli heat-stable enterotoxin (ST) appears to cause intestinal fluid secretion by activating intestinal particulate guanylate cyclase. Recent studies suggest that chlorpromazine and quinacrine reduce the intestinal secretory response to ST and activation of guanylate cyclase by ST. We have examined the effects of lanthanum chloride, another agent that has been shown to inhibit calciu...

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Reduction of the secretory response to Escherichia coli heat-stable enterotoxin by thiol and disulfide compounds.

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The cystic fibrosis (CF) transmembrane conductance regulator (CFTR) is an important pathway for duodenal mucosal bicarbonate secretion. Duodenal biopsies from CF patients secrete bicarbonate in response to heat-stable enterotoxin from Escherichia coli (STa) but not cAMP. To explore the mechanism of STa-induced bicarbonate secretion in CF more fully, we examined the role of CFTR in STa-stimulate...

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Properties of synthetically produced Escherichia coli heat-stable enterotoxin.

The properties of a synthetically produced peptide composed of the same primary structure of 18 amino acids described for human Escherichia coli heat-stable enterotoxin were compared with those of purified heat-stable toxin obtained by bacterial growth. The dosage required to evoke fluid secretion in the suckling mouse and rat ligated ileal loop assays was the same for both toxins. The antigeni...

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ژورنال

عنوان ژورنال: Infection and Immunity

سال: 1982

ISSN: 0019-9567,1098-5522

DOI: 10.1128/iai.35.2.483-488.1982